sensory nerve

'This is the first time we have been able to use synovial fluid from human osteoarthritis patients to excite sensory nerve cells, making it more clinically-relevant than mouse studies alone, and so will hopefully help translating treatments from bench to bedside,' says Dr Ewan St John Smith from the Department of Pharmacology at the University of Cambridge.

Table: Motor nerve conduction velocity (MNCV) and sensory nerve conduction velocity (SNCV) data of the patient.

Sensory nerve action potential was recorded antidromically using ring electrodes.

Impact of long versus short duration diabetes on sensory nerve conduction velocity in Type 2 diabetes mellitus.

We suggest that mast cell degranulation plays a key role in vasodilation and vascular permeability associated with activation of trigeminal sensory nerve fibers in the dura mater where is the origin of migraine pain.

The stimulation intensity was 10-30 mA for sensory nerve conduction studies and 10-50 mA for motor nerve conduction studies.

Previously, we found that the sensory nerve tract preimplanted in the tissue-engineered bone (TEB) could significantly improve osteogenesis of the TEB [4, 5], but the underlying mechanism was still largely unknown.

Whilst recent studies have established good diagnostic utility and reproducibility for sensory nerve evaluation in the skin and cornea, there has been limited application of these techniques in the clinic [3].

In a sensory nerve conduction study (NCS), we attempted to derive sensory nerve action potentials (SNAPs) of the thumb, index, and middle fingers.

Antidromic sensory nerve action potentials (SNAP) for median and ulnar sensory nerves were recorded with standard ring electrodes.

The sensory nerve action potential (SNAP) was unable to be obtained using the left superficial peroneal sensory nerve conduction study.

It involves cutting the original sensory nerve cells out of the root and implanting the remaining root directly into a deeper structure in the spinal cord.

Compound muscle action potential (CMAP) (normal > 6.8 mV), motor nerve conduction velocity (M-NCV) (normal > 49.4 m/sec), motor distal latency (normal < 3.8 msec), sensory nerve action potential (SNAP) (normal > 10 [micro]V), and sensory nerve conduction velocity (SNCV) (normal > 40.4 m/sec) were measured.

Previous study had said that the monofilament testing can be one of the valid and standard screening tests for sensory nerve function assessment.

The electroneuronographic studies of 11 and 25 November 2014 showed significantly decreased compound muscle action potential (CMAP) amplitudes and preserved sensory nerve action potential (SNAP) amplitudes, with normal distal latencies and conduction velocities, favouring a diagnosis of an AMAN variant of the Guillain-Barre syndrome (Tables 2 and 3).