rickets


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Related to rickets: hypophosphatemic rickets
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Synonyms for rickets

childhood disease caused by deficiency of vitamin D and sunlight associated with impaired metabolism of calcium and phosphorus

References in periodicals archive ?
Rickets was common in the past, but it mostly disappeared in the Western world during the early 20th century after foods were fortified with vitamin D.
Severe cases of rickets may result in stunted growth and skeletal deformities in children.
Rickets can also be caused by a genetic abnormality that does not allow Vitamin D to be absorbed correctly.
Clinically, VDDR1A is characterized by hypotonia, muscle weakness, inability to walk, growth failure and radiographic findings of rickets. Typical laboratory findings are hypocalcaemia, elevated serum levels of alkaline phosphatase (ALP) and of PTH with low or normal levels of 1,25-[OH.sub.2]D despite normal or increased concentrations of 25-OHD (9,13).
There were an additional 474 admissions in 2017/18 where the main or secondary reason was rickets, up from 445 the year before.
Her youngest sister was born in Limehouse and was a very poorly baby who also had rickets.
The 13 diseases are gout, tuberculosis, malnutrition, whooping cough, measles, scurvy, typhoid, scarlet fever, diphtheria, mumps, rickets, cholera, and vitamin D deficiency.
Exclusive figures from NHS Digital show there were at least 655 occasions in 2017/18 on which people were treated as inpatients in hospitals in the area for one of 13 diseases common over 100 years ago: gout, tuberculosis, malnutrition, whooping cough, measles, scurvy, typhoid, scarlet fever, diphtheria, mumps, rickets, cholera, and vitamin D deficiency.
Vitamin D Resistant Rickets (VDRR) is also known as X-linked hypophosphatemia with a prevalence of 1:20.000 people (1-4).
The girl was suffering from malnutrition and had developed rickets.
It would be recalled that the Member of Parliament (MP) for the Cape Coast South Constituency, Mr Kweku Rickets Hagan, already has two buses that are used for a similar purpose.
THURSDAY, May 24, 2018 (HealthDay News) -- For children with X-linked hypophosphatemia, subcutaneous burosumab is associated with decreases in rickets severity and with improved renal tubular phosphate reabsorption, according to a study published in the May 24 issue of the New England Journal of Medicine.
Recent studies revealed that the gene responsible for the regulation of phosphate levels is located on the X chromosome, and mutations in this gene result in hypophosphatemic rickets (3).
This article explores the role of infant and child diet in the manifestation of vitamin D deficiency, called rickets, in non-adults (<18 years) from a post-medieval cemetery called Middenbeemster, in rural Holland.