Clinical profile and opportunistic infection
in HIV/-AIDS patients attending S.S.
Conclusion: Tuberculosis in HIV-AIDS patients is still an important and fairly common opportunistic infection
. All HIV-AIDS patients needs to be screened for evidence of TB and all the patients with TB needs to be screened for HIV co-infection.
Generally, opportunistic infections
result from at least 1 of 3 mechanisms: (1) external acquisition of a virulent pathogen (e.g., meningococcal meningitis or pneumococcal pneumonia), (2) reactivation of an internal latent organism (e.g., herpes simplex virus, herpes zoster virus [shingles], or TB, and (3) invasion of a normally commensal organism (e.g., bacteria, viruses, fungi, or protozoa/ parasites).
(10) We could not find any published literature regarding kala-azar as an opportunistic infection
exclusively in children with HIV infection.
separately accounts for 106 of 249 deaths, which is 42.57% representing the most common cause of death among the HIV related deaths in this study (Figure 6).
Patients who had active opportunistic infections
were treated and put on Anti-Retroviral Therapy as per protocol.
 It has been observed that the clinical course of HIV infection varies considerably from patient to patient and the spectrum of opportunistic infections
also varies depending on the geographic regions.
Nontuberculous mycobacteria, which are widespread in the environment, frequently cause opportunistic infections
in immunocompromised patients.
Spectrum of opportunistic Infections
and profiles of CD cell counts among AIDS patients in Northern India Infection 2003, 31, 336-40.
Starting in 1996, several studies and cohorts demonstrated dramatic decreases in mortality and opportunistic infections
with the widespread availability of the protease inhibitors.
Also, the observed gender differences in viral load were not significantly associated with HIV disease progression based on time to death or with development of an AIDS-defining opportunistic infection
The fourth major opportunistic infection
for HIV-infected patients, Toxoplasma gondii, is also prevented with trimetho-prim-sulfamethoxazole.
Sometime just after opportunistic infection
rates plummeted with the widespread use of potent antiretroviral therapy (1996-1997) and just before federal funding for HIV/AIDS began to wither (circa 2002), the AIDS community began to feel a sense of uneasiness.
Current therapies for microsporidiosis, a serious opportunistic infection
in persons with AIDS, organ transplant recipients, children, travelers, contact lens wearers, and the elderly, were reviewed.
The body's defenses are suppressed, exposing patients to opportunistic infection
by harmful invaders.