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Related to nociceptive: allodynia, nociceptive reflex
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Words related to nociceptive

caused by or in response to pain

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Scientists previously thought that the cells called nociceptive fibers were where pain begins.A
This non-randomized, control trial study aimed to assess hemodynamic responses to nociceptive stimuli, when Dexmedetomidine is used as an adjuvant anaesthetic to isoflurane in paediatric patients undergoing video laparoscopic surgeries.
As assessed by the hot plate test, EOPPgfl significantly increased the latency time for nociceptive response to the thermal stimulus starting at 30 min and spanning throughout the observation period of 180 min.
Medication overuse could increase the cerebral cortex and trigeminal neuronal excitability, which would make migraine brain easy to suffer from cortical depression and generate central and peripheral sensitization by the trigeminal system.[29] A previous study also confirmed the sensitization of the trigeminal nociceptive neurons[30] and pain facilitation of the trigeminal nociceptive systems[31] in MOH.
The purpose of this study was to identify environmental nociceptive stimuli that could impact recovery of children who experience PSH after brain injury.
In addition, recent scientific evidence indicate that those pain-related brain regions, which were previously included in the "pain neuromatrix," are, in fact, not exclusively engaged in the processing of nociceptive information but also in the detection of other sensory stimuli [37].
In each session, TMS and thermal nociceptive stimulation intensities were determined (see below), and four randomized blocks of 10 MEPs were recorded: two blocks with (pain condition) and two blocks without (control condition) thermal nociceptive stimulation, for a total of 20 MEPs per condition.
VGSCs in primary nociceptive neurons of the TG are involved in pain transduction and transmission processes in orofacial regions [16-18].
On the other hand, TMD can be considered a potential perpetuating factor for migraine because it acts as a constant nociceptive input that contributes to maintaining central sensitization and abnormal pain processes."
All these components have since been mapped throughout the peripheral nerve terminals and extend up to the supraspinal centers, which constitute the pain (nociceptive) pathway (3).
[8] The venom-induced vasosensory reflex responses lasts longer than the other nociceptive agonists (capsaicin/anandamide/[alpha] [beta]Me-ATP)-induced responses.
Nociceptive processes in craniofacial muscles are believed to play a role in the development and maintenance of TTH.
Translocation is necessary to allow phosporylation by PKC[epsilon] of specific cellular targets located in the plasma membrane in nociceptive neurons [16,17], including TRPV1 (Transient Receptor Potential Vanilloid 1) ion channels [18] and several other targets involved in nociceptor sensitization and excitability.