However, we found no evidence that DDTs reduce exercise or cause hyperphagia
(Howell et al.
chevrieri would show the post-fast hyperphagia
phenomenon after re-feeding.
Energy density in cafeteria diet induced hyperphagia
in the rat.
Effects of palatability-induced hyperphagia
and food restriction on mRNA levels of neuropeptide Y in the arcuate nucleus.
Although scientific evidence has shown the neurobiological basis of the behavioral changes and patterns of hyperphagia
, interventions which involve giving guidance to parents and caregivers in respect of parenting practices and the management of the children's behavioral problems are considered to be a priority; previous studies report that these interventions can not only improve a range of behavioral indicators in the children but also help support the caregiver in relation to the emotional difficulties they face [16-21].
Decreasing hypothalamic insulin receptors causes hyperphagia
and insulin resistance in rats.
Cinnamaldehyde supplementation prevents fasting-induced hyperphagia
, lipid accumulation, and inflammation in high-fat diet-fed mice.
However, with the drug, the two patients showed a "dramatic alteration in their sensation of hunger, and their hyperphagia
disappeared for the first time in their lives, leading to a normalization of their satiety," said first author Dr.
One of them was with hyperphagia
and four with anorexia.
Prader-Willi syndrome is a genetic disorder characterized by hypotonia, obesity, short stature, mental retardation, hyperphagia
, hypogonadism and low life expectancy.
The clinical presentation is characterized by hypotonia, hyperphagia
, mental retardation, and dysmorphism.
3 In another report, liraglutide was found to be effective in two obese patients with hypothalamic hyperphagia
and MRI- detectable hypothalamic lesions.
The endocrine, autonomic, and behavioral problems such as hyperphagia
and obesity, psychomotor retardation, emotional immaturity, apathy, short term memory deficits, and incontinence are produced by the thalamus and frontal lobes correspondingly.
Moreover, this steroid is essential for the development of insulin resistance, for the alterations in muscle function, and for the alterations in bone growth and hyperphagia
these animals manifest .
Peripheral oxytocin activates vagal afferent neurons to suppress feeding in normal and leptin-resistant mice: a route for ameliorating hyperphagia