of substances (endogenous/exogenous) is manipulated by different mechanisms i.e.
There was no correlation between urinary pH and electrolyte excretion
. Sulfur excretion
was correlated only with [Mg.sup.2+] excretion
([R.sup.2=0.55,P<] 0.001), and this demonstrated the high correlation with [Na.sup.+] ([R.sup.2=0.74,P<] 0.001), [K.sup.+] ([R.sup.2=074,P<0001]), phosphorus ([R.sup.2=072,P<] 0.001) and [Cl.sup.-] excretion
([R.sup.2=070,P<] 0.001) (Table 4).
A 24-h urine collection revealed a urinary creatinine excretion
of 1160 mg.
"These results suggest that low urine ammonium excretion
identifies individuals at high risk of chronic kidney disease progression or death irrespective of the serum bicarbonate concentration," said Kalani Raphael from the University of Utah in the US.
per nephron = urinary phosphate excretion/nephron number [varies] FGF23 (1)
The 24-hour urinary sodium and potassium excretion
values were used as surrogates for the daily sodium and potassium intake.
We hereby aimed to study the effects of the SGLT2 inhibitor empagliflozin in healthy volunteers in artificially induced SIADH with focus on urinary volume excretion
, glucosuria, and change of serum sodium level.
Preliminary results showed that the three patients in the first cohort who received monthly subcutaneous doses of lumasiran at 1 mg/kg for three months experienced greater than 50% decreases in urinary oxalate excretion
relative to baseline.
On the basis of this evidence, in this work, we analyzed the urinary excretion
of AQP1, AQP2, and AQP5 in three groups of patients: 12 diabetic with no sign of nephropathy (DM), 12 diabetic with histologic diagnosis of diabetic nephropathy (DN), and 11 diabetic with nondiabetic nephropathy (NDN).
In clinical practice, the analysis of fractional excretion
(FE) as a substitute of tubular dysfunction in the CKD patients was restricted to phosphate metabolism so far .
It is intuitive to speculate that the excretion
of 145 kDa Rituximab into the urine due to nonselective large molecular size proteinuria might contribute to this clinical important issue.
To date, to the best of our knowledge, no data exist evaluating compensatory ammonium excretion
in the context of postoperative acidosis of either origin after CABG and cardiac surgery in general.
Our murine data established a PKU bone phenotype characterized by decreased BMD and defective bone biomechanical performance, which was worsened by a low-Phe amino acid (AA) diet in association with increased renal net acid and calcium excretion
and increased renal mass [8-10].
Urinary calcium excretion
varies for geographic areas.