Levin & Baltaxe analyzed 50 patients with celiac stenosis due to arcuate ligament compression and/or celiac plexus with complete absence of abdominal symptoms or referring to the gastrointestinal tract, submitted to lateral aortogram by other clinical causes.
After several studies reported the success of the surgical treatment by sectioning the arcuate ligament and/or the celiac plexus (Terpstra; Marable et al.
1966), several authors reported the celiac plexus compression by both agents--the median arcuate ligament and the celiac axis (Rob, 1966; Stoney & Wylie; Bobbio et al.
Based on this clinical report, many authors agreed with this defined clinical entity, considering the celiac plexus as the etiologic agent in the celiac trunk compression syndrome, which is formed by an abundant neurofibrous tissue able to constrict this vessel (Drapanas & Bron, 1966; Debray et al.
We agree with those authors who mentioned that during the surgical revascularization process of the celiac trunk, compressed by the median arcuate ligament, the section or exerese of the rich fibronervous celiac plexus, whose fibers are deep intermingled with those of the median arcuate ligament, to access this ligament is almost a need (Harjola & Lahtiharju; Balmes et al.