catalepsy


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Related to catalepsy: narcolepsy
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  • noun

Words related to catalepsy

a trancelike state with loss of voluntary motion and failure to react to stimuli

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Catalepsy was produced by injection haloperidol 1 mg/kg intraperitoneally half an hour after the pretreatment with test drug or half an hour after pretreatment with vehicle (distilled water) for control.
Figure 5 shows the effects of rotenone, curcumin and their co-administration on the induction of catalepsy. Statistical analysis by one-way ANOVA revealed significant effects of treatment on % cataleptic score (F=143.66, p<0.01).
94 (a) 3 3.64 [+ or -] 0.65 8.13 [+ or -] 1.18 (a) 4 3.69 [+ or -] 0.57 11.75 [+ or -] 1.08 (a) Catalepsy bar test 0 2.50 [+ or -] 1.17 2.17 [+ or -] 1.27 1 2.67 [+ or -] 1.15 5.00 [+ or -] 1.86 (a) 2 2.75 [+ or -] 0.87 11.25 [+ or -] 2.26 (a) 3 2.92 [+ or -] 1.083 20.92 [+ or -] 2.61 (a) 4 2.92 [+ or -] 1.31 27.50 [+ or -] 3.75 (a) Adhesive Removal Test 0 11.92 [+ or -] 1.24 12.17 [+ or -] 1.53 1 12.25 [+ or -] 1.48 15.75 [+ or -] 1.82 (a) 2 12.58 [+ or -] 1.51 21.75 [+ or -] 2.14 (a) 3 12.58 [+ or -] 1.09 25.67 [+ or -] 4.83 (a) 4 12.75 [+ or -] 1.29 35.75 [+ or -] 2.60 (a) Manual gait analysis 0 4.00 [+ or -] 0.
Medical explanations for zombification include catalepsy and other psychiatric disorders, residual effects from neurotoxins, drug toxicity, near death in a location with limited medical care, and a mistaken belief of death.
For this purpose, apomorphine-induced climbing, haloperidol-induced catalepsy, and MK-801-induced hyperlocomotion tests were used.
News.com.au reported catalepsy might be the reason for Jimenez's condition.&nbsp;(https://www.ncbi.nlm.nih.gov/mesh/68002375) Catalepsy , according to the&nbsp;National Center for Biotechnology Information, is a condition where the body goes into a state of inactivity and has decreased responsiveness to any sort of stimuli.
There should be 3 or more of the following 12 findings for the diagnosis of catatonia (4) Stupor (no psychomotor activity, not actively relating to the environment) Catalepsy (passive induction of a posture held against gravity) Waxy flexibility (resistance to positioning by the examiner) Mutism (no or very little verbal response) Negativism (no response to external stimuli) Posturing (maintenance of a posture against gravity) Mannerism (caricature of normal actions) Stereotype (abnormally frequent and repetitive, non-goal-directed movements) Agitation Grimacing Echolalia (mimicking other's speech) Echopraxia (mimicking other's movements)
Various agonists of CB1 and CB2 receptors prevent or attenuate symptoms of CINP in animal models; however, various problems are associated with CB1 receptor activation including physical dependence, development of tolerance, and other cannabimimetic adverse effects such as hypothermia and catalepsy. Options to circumvent these problems that have been explored include the use of a CB1 receptor PAM or CB2 receptor-selective agonists.
A DSM-5 [7] diagnosis of catatonia requires the presence of three or more of the following symptoms: stupor, catalepsy, waxy flexibility, mutism, negativism, posturing, mannerism, stereotypy, agitation, grimacing, echolalia, and echopraxia.
Kuwana, "Actions of adenosine A2A receptor antagonist KW-6002 on drug-induced catalepsy and hypokinesia caused by reserpine or MPTP," Psychopharmacology, vol.
Motor changes: Relaxation, weakness, flask-spastic paralysis, catalepsy, hyperkinesis, increase in muscle strength.
Acute or chronic administration of the dopamine D1-like antagonist (SCH23390) caused catalepsy in rats [29, 30], while administration of dopamine D1-like agonists (SKF) caused stimulation of locomotor activity [31].