atherogenesis


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Related to atherogenesis: dyslipidemia
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  • noun

Words related to atherogenesis

the formation of atheromas on the walls of the arteries as in atherosclerosis

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This establishes 5'-nucleotidase-derived adenosine as a direct or indirect regulator of atherogenesis "as discussed elsewhere (Boring, Y.
The paracrine or vasocrine secretion of epicardial inflammatory adipokines, such as tumor necrosis factor alpha, plasminogen activator inhibitor-1, interleukin-6, interleukin -1b, monocyte chemo-attractant protein-1, and resistin contribute to the metabolic and inflammatory milieu that promotes atherogenesis (43).
The vascular biology of nitric oxide and its role in atherogenesis.
Both Th1 T cells and macrophages are known long-term reservoirs for HIV [33] and thus it is likely that Th1 T cell and macrophage cross-talk within the coronary circulation, in the presence of a dysfunctional endothelium, can contribute to atherogenesis (Figure 1).
25,26) Thus, the role of vitamin D in inflammatory mediated atherogenesis requires further investigation.
It is important that plaque stability and the influence on atherogenesis are also studied in these trials," said Isermann.
They found that individuals with periodontal disease whose biomarkers showed increased bacterial exposure were more likely to develop coronary heart disease or atherogenesis (plaque formation in the arteries).
Maternal hypocholesterolemia has been reported to enhance early atherogenesis and marked increase in fatty streaks in human fetuses [3].
4,6) Accelerated atherogenesis is often suggested, but has not been proven.
20) Recently released follow-up data from the APPROVe trial further establishes that Cox-2 inhibition contributes to atherosclerotic progression and atherogenesis, and that the hazard continues even after the drug is discontinued.
In a manner analogous to atherogenesis, the cellular components of the vessel wall react to barotrauma and foreign body application with a broad array of responses.
Screening for an enlarged waist in combination with elevated triglycerides appears to be a simple, effective tool to flag postmenopausal women at increased risk for accelerated atherogenesis and related adverse outcomes, reported Laszlo B.
Experimental animal studies have shown that bacterial and viral agents could contribute to atherogenesis.
The expression of endothelial adhesion molecules promotes the attachment of leukocytes and subsequently leads to atherogenesis.