angiotensin II

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Words related to angiotensin II

a potent vasopressor agent formed from angiotensin I

References in periodicals archive ?
We found that angiotensin II is an effective intervention for these patients, significantly increasing blood pressure in this life-threatening situation.
The comperative pharmacology of angiotensin II receptor antagonist.
The report provides a snapshot of the global therapeutic landscape for Type-2 Angiotensin II Receptor (Angiotensin II Type-2 Receptor or AT2 or AGTR2)
Raij, "Skeletal muscle insulin resistance in salt-sensitive hypertension: role of angiotensin II activation of NFxB," Cardiovascular Diabetology, vol.
Catt, "Differential distribution of at1 and at2 angiotensin II receptor subtypes in the rat brain during development," Proceedings of the National Academy of Sciences of the United States of America, vol.
Cultivation of endothelial progenitor cells with angiotensin II significantly increased the number of [beta]-galactosidase-positive cells (52.
Angiotensin II binds to specific receptors to initiate a vast array of signaling pathways and physiological responses.
sup][1] Angiotensin II (Ang II), the major effector of RAS, [sup][2] exerts its biological effects mainly through specific receptors, namely angiotensin type 1 receptor (AT [sub]1 R) and type 2 receptor.
An angiotensin-converting-enzyme (ACE) inhibitor, OPtainl20 suppresses production of angiotensin II, which decreases the peripheral resistance and blood volume, thereby reducing blood pressure.
Drug type Options How it works Diuretic Hydrochloro Promotes elimination of sodium thiazide and water, reducing blood volume (Microzide) ACE inhibitor Lisinopril Blocks production of angiotensin II, (Prinivil) a chemical that constricts blood vessels Angiotensin II Candesartan Limits the action of angiotensin II receptor (Atacand) blocker (ARB) Calcium channel Amlodipine Relaxes the blood vessels blocker (Norvasc)
The latter two are a part of the renal vasodilator system, whereas the renin-angiotensin system represents the renal vasoconstrictor, with the vasodilator systems modulating vasoconstrictive effects of angiotensin II on one hand, and angiotensin II stimulating the secretion of vasodilatory components on the other, which prevents substantial increase in the renal vascular resistance.
Conclusion: Inhibition of RAS by converting enzyme inhibitors or angiotensin II receptor blockers captopril, enalapril, valsartan or telmisartan produce beneficial effects on the markers of metabolic syndrome and can reduce the frequency of metabolic syndrome in type 2 diabetic patients.
Interestingly, the force with which the heart ejected blood remained unchanged with the additional stressor, angiotensin II, a powerful constrictor of blood vessels.
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