amygdaloid nucleus

The MePD is important for higher-level, supramedullary control of cardiovascular functions (6-8), and, in this sense, sends projections to the central amygdaloid nucleus (CeA) and then to the A1 noradrenergic cells of the caudal ventrolateral medulla and the nucleus of the solitary tract (NTS; 9,10).

Magnetic resonance (MR) imaging showed symmetrical calcifications on the amygdaloid nucleus and uncus.

Anatomical interactions between the central amygdaloid nucleus and the hypothalamic paraventricular nucleus of the rat: A dual tract-tracing analysis.

[11-13] The central amygdaloid nucleus receives afferents projection from the basolateral complex and has projections to the hypothalamus and brainstem; these connections suggest that it is the final common pathway for the fear responses.

As well as being interrelated to vegetative activity and motor coordination of talking and deglution, it has also been attributed with regulating blood-pressure, controlling visceral pain, thermal regulation and motivational-affective responses to pain due to its connections with the solitary nucleus, raphe magnus, parabrachial area and amygdaloid nucleus (trigeminal-parabrachial-amygdaloid ipsilateral tract) with its usual visceral, nutritional (anorexia), respiratory, thermal and inhibitory descending pain effects (Caous et al.; Armstrong & Holpkins; Jasmin et al., 1997; Paues et al., 2006; Rybka & McCulloch, 2006).