periodontitis

Neutrophil-mediated tissue injury in periodontal disease pathogenesis: findings from localized aggressive periodontitis.

Keywords: Interleukin-6; Interleukin-10; Polymorphism, Genetic; Aggressive Periodontitis.

Interleukin-1 and tumor necrosis factor-alpha gene polymorphisms in Turkish patients with localized aggressive periodontitis.

Enhanced accumulation of neutrophils in the pocket epithelium and adjacent connective tissue of patients with chronic periodontitis and with generalized aggressive periodontitis (GAP)s was associated with the upregulation of IL8, intercellular adhesion molecule-1, IL-1b and TNF--a expression, which related to the severity and activity of generalized aggressive periodontitis.

Effect of azithromycin, as an adjunct to nonsurgical periodontal treatment, on microbiological parameters and gingival crevicular fluid biomarkers in generalized aggressive periodontitis," Journal of Periodontal Research, vol.

Sub gingival Plaque samples were taken with curettes from Chronic periodontitis patients in deep CAL >6mm and shallow pockets CAL>3mm in same patients and from aggressive periodontitis in CAL>6mm.

The authors indicated, however, more research is needed to determine effectiveness of PDT in medically compromised patients and aggressive periodontitis, as well as in treatment of peri-implant disease and during supportive periodontal therapy.

Demographic, clinical, and microbial aspects of chronic and aggressive periodontitis in Colombia: a multicenter study.

sup][1],[2] Unlike the most common form of chronic periodontitis (CP), aggressive periodontitis (AgP) develops early in life, progresses faster, and presents more destructive clinical features.

In their study, in aggressive periodontitis patients group, 50% of the sites were having detectable levels of RANKL, compared with 46.

Aggressive periodontitis was formerly known as juvenile periodontitis or localized early-onset periodontitis.

Diagnosis and Treatment Alternatives for Aggressive Periodontitis or as a Manifestation of Systemic Disease in 3-to-12-year-old Children.

There is also a case report by Seremidi and co-authors describing the therapeutic management of an 8-year-old child with generalised aggressive periodontitis.

Initially some researchers3,4 considered HMS a variant of Papillon Lefe'vre syndrome (PLS) on the basis of common clinical features such as palmo-plantar keratoderma (abnormal hyperkeratosis of the palms and soles) and aggressive periodontitis.

Susceptibility to amoxycillin of anaerobic bacteria in patients with aggressive periodontitis