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  • noun

Antonyms for hyponatremia

abnormally low level of sodium in the blood

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(1) Biochemical severity (a) Mild hyponatremia, 130-135 mmol/L (b) Moderate hyponatremia, 125-129 mmol/L (c) Profound hyponatremia, <125 mmol/L (2) Time of onset (a) Acute hyponatremia <48 hours (b) Chronic hyponatremia >48 hours (3) Symptoms (a) Symptomatic hyponatremia (b) Asymptomatic hyponatremia (4) Volume status (a) Hypovolaemia (b) Normovolaemia (c) Hypervolaemia (5) Serum osmolality (a) Hypotonic hyponatremia, <275 mOsm/kg (b) Isotonic hyponatremia, 275-295 mOsm/kg (c) Hypertonic hyponatremia, >295 mOsm/kg Table 4: Assessment of volume status.
When acute hyponatremia was developed (over 48 hours or less) it should be treated by administering hypertonic 3% saline to achieve a mildly hyponatremic level.
Acute hyponatremia and seizures in an infant after a swimming lesson.
When we realized that the neurologic sequelae had been caused by acute hyponatremia, we infused a 3% sodium chloride solution until the neurologic symptoms resolved, at which time the patient's serum sodium level had risen back to 135 mEq/L.
In addition, the rapid correction of acute hyponatremia can induce central potine myelinolysis, which emphasizes the importance of preventing hyponatremia (Hassan, Kruer, & Fuhrman, 2007).
Hospital-acquired acute hyponatremia and reports of pediatric deaths
In acute hyponatremia the most important risk to address is cerebral edema, because brain cells have too little time to adapt to cell swelling.
Any athlete exhibiting signs and symptoms of acute hyponatremia listed above should be screened for EAH by measuring plasma or serum [Na+].
Acute hyponatremia with resultant transcellular fluid shifts and cerebral edeme has been postulated as the primary cause.
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