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The use of autologous activated macrophages for nerve regeneration is based on technology developed in the laboratory of Prof.
Furthermore, sustained overproduction of nitric oxide (NO) generated from inducible nitric oxide synthase (iNOS) expressed in activated macrophages and endothelium, can lead to a modulation of leukocyte infiltration, a cyclo-oxygenase-2 (COX-2)-dependent production of inflammatory prostaglandins (PGs), and an interaction of NO with leukocyte-derived [O.
Various plant-derived bioactive molecules are thought to reduce the inflammatory response of activated macrophages (Mamani-Matsuda et al.
HMGB1 is a ubiquitous nuclear protein that can be released by any damaged cell or by activated macrophages and certain other cell types.
Downregulating TNF-[alpha] expression in activated macrophages in response to Ti particles, while simultaneously upregulating BMP-2 expression to promote osteoblast differentiation, is expected to be effective in the treatment or prevention of periprosthetic ostelysis.
YKL-40 promotes maturation of monocytes to macrophages; afterwards YKL-40 gets secreted by macrophages during the late stages of differentiation and eventually by the activated macrophages (12-15).
Galectin-3 is expressed by activated macrophages and induces cardiac fibroblasts to proliferate and deposit type I collagen in the myocardium (1).
111) These data suggest that alternatively activated macrophages are strongly associated with multiple forms of lung fibrosis and may serve as targets for therapeutic intervention.
Hollzmuller et al (19), demonstrated that natural resistance to Leishmania infection depends on intracellular parasite killing by activated macrophages through production of L-arginine-nitric oxide metabolic pathway.
Chemokines and activated macrophages in HIV gp120-induced neuronal apoptosis.
During this process, the activated macrophages would release cytokines such as TNF-[alpha], IL-1[beta], IL-6 and IFN-[gamma] to inhibit the growth of a wide variety of tumor cells and microorganisms with the help of NO cytolytic function and iNOS (Higuchi et al.
Clinical studies have demonstrated that women with endometriosis have increased volume of peritoneal fluid, increased concentration of activated macrophages, and increased peritoneal concentrations of prostaglandins, interleukin-1, tumor necrosis factor, and proteases.
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