hypercalciuria

In absorptive hypercalciuria, the primary defect is in the intestine, which causes a transient increase in plasma calcium; the kidneys get rid of the excess calcium.

Preminger treats most of his absorptive hypercalciuria patients with a thiazide and diet.

Sodium cellulose phosphate, introduced in the early 1980s, is a calcium-ion-binding resin that binds to calcium in the gut during meals, preventing calcium absorption, the underlying defect in patients with absorptive hypercalciuria.

Evidence for normal vitamin D receptor messenger ribonucleic acid and genotype in absorptive hypercalciuria.

Evidence for disordered control of 1,25-dihydroxyvitamin D production in absorptive hypercalciuria.