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infection caused by parasites transmitted to humans from infected cats

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6%, respectively were calculated considering cPCR and clinical diagnosis (146 samples, which included 94 patients with cerebral toxoplasmosis, 10 newborns with congenital infection, 21 patients with ocular toxoplasmosis, six pregnant women with toxoplasmosis, and 15 postmortem patients with severe disseminated toxoplasmosis) (Table 2).
Ocular toxoplasmosis past, present and new aspects of an old disease.
Ocular toxoplasmosis may cause symptomatic progressive chorioretinitis.
Anti-Toxoplasma gondii antibodies in serum and aqueous humor in patients with ocular toxoplasmosis retinal lesions.
3) In the United States, for instance, the proportion of infected patients who had ocular toxoplasmosis is approximately 2%, but the prevalence of ocular involvement is greater in other parts of the world such as Brazil, where it is around 18%.
Conclusions: The major causes of blindness in children attending the "Instituto para ninos ciegos y sordos del Valle del Cauca," are preventable blindness with primary prevention the retinopathy of prematurity and ocular toxoplasmosis, or with secondary prevention retinopathy of prematurity, glaucoma, retinoblastoma and congenital cataract.
Damms T, Bohnkc M, Behrend Serdin B: Result of therapy in ocular toxoplasmosis.
Whether the increased prevalence and severity of ocular toxoplasmosis in Brazil are attributable to host or parasite genetic factors or differences in exposure rate is uncertain.
16) In an animal model of ocular toxoplasmosis, treatment with anti-TNF-[alpha] antibodies resulted in worsening of ocular lesions, with the development of retinal necrosis and marked inflammation of the vitreous and underlying choroid.
The city of Erechim, Brazil, has a 17% prevalence of ocular toxoplasmosis, and type 1 Toxoplasma gondii predominates.
This finding is consistent with reports of vertical transmission of the infection as a result of ocular toxoplasmosis reactivation.
gondii strains isolated from patients with ocular toxoplasmosis in Brazil were highly divergent when compared to the previously described clonal lineages from North America and Europe, suggesting more frequent sexual recombinants resulting in mixed genotypes.
Besides the seven initial cases of retinochoroiditis that occurred during acute disease, another two patients acquired late retinal lesions, developed at least two years after the primary infection, which increases the prevalence of postnatal-acquired ocular toxoplasmosis to 3.