cardiac muscle

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Related to Cardiomyocyte: cardiac muscle
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Synonyms for cardiac muscle

References in periodicals archive ?
Cardiomyocyte hypertrophy induced by the TLR 2/4 agonists was measured by calculating cardiomyocyte surface area.
Citation: Mitch Biermann et al., Epigenetic Priming of Human Pluripotent Stem Cell-Derived Cardiac Progenitor Cells Accelerates Cardiomyocyte Maturation.
Once the heart is fully formed, the cells that make up heart muscle, known as cardiomyocytes, have very limited ability to reproduce themselves.
For H9c2 cells, the SI-3 h treatment caused mild cardiomyocyte injury in models 1, 2, and 3, as demonstrated by more granule generation in cytoplasm [Supplemental Figure 2 [SUPPORTING:2]]a.
Briefly, cell culture medium including (90 min) or excluding (24 h) [Ca.sup.2+] dye was replaced with 25 [micro]l of staining solution (2 [micro]lg/ml Hoechst 33342 and 0.2 [micro]lM MitoTracker Orange in iCell cardiomyocyte maintenance medium).
A critical regulator of cardiomyocyte contractibility is calcium ([Ca.sup.2+]) mobilization.
miRNA-1, contributing to ~40% of total miRNAs in the heart, has been shown to promote cardiomyocyte proliferation and suppress apoptosis during development; however, its function in cardiac reprogramming remains unknown [33].
showed that deletion of YAP in mice containing the Nkx2.5-Cre resulted in reduced cardiomyocyte proliferative ability and eventually led to embryonic death at stage 10.5.
What are challenges in biological repair of heart?: The real challenge is to gain insights of dynamics of fetal to adult cardiomyocyte gene expression encoding proteomics for a well-organized sarcomeres, t-tubules, and ion channels to ensure an endogenous repair of adult heart.
BDNF Reduced Cardiomyocyte Apoptosis after Septic Shock That Was Reduced by L-NAME.
Considering the similarities in the pathologic mechanisms underlying ischemia/reperfusion injury between cardiomyocyte and intestinal, we question if an antiapoptotic mechanism is also involved in the cardioprotective effect of oleuropein.
However, the family of miR-15 has negative effect on cardiomyocyte proliferation [53, 54].
This is particularly important in modeling of diseases caused by malfunctioning of potassium channels, as different types of potassium channels play a key role in cardiomyocyte repolarization in different species [1].
These cells are called cardiomyocytes, and losing them puts people at risk of heart failure - a condition wherein the heart cannot pump blood effectively to the rest of the body.