Klf15, in turn, controls the level of a potassium channel-interacting protein (KChIP2), which affects how potassium flows out of heart muscle cells called cardiac myocytes
TNF-[alpha] has been shown to drive production of manganese superoxide dismutase , a free radical scavenger, as well as certain heat shock proteins  in adult mammalian and feline cardiac myocytes
(12) In this study, we use the rat coronary artery ligation model to assess the effects of estrogen on indices of left ventricular remodeling, including cardiac myocyte
Using a guinea pig model, Shiba et al . reported that transplanted cardiac myocytes
were able to form gap junctions with the surrounding host myocardium and achieve 1:1 host-graft coupling.
Mochly-Rosen, "A selective [epsilon]-protein kinase C antagonist inhibits protection of cardiac myocytes
from hypoxia-induced cell death," Journal of Biological Chemistry, vol.
The recent study indicated that NNBP enhanced myocardial perfusion, depressed cardiac myocyte
apoptosis, decreased myocardial stunning, and improved heart contractile function in comparison with traditional NHCA.
showed recently that short pretreatment with NO donor (NaN[O.sub.2]) combined with hypoxia protects neonatal cardiac myocyte
but not cardiac fibroblast from hypoxic injury, and apoptosis decreased in human ES-derived cardiac myocytes
Additionally, modulation of electrical activity of cardiac myocyte
by natural occurring chemical compounds may present a therapeutic application, especially in the context of cardiac arrhythmias (Li et al.
This is logical when one considers the nature of the acute coronarysyndrome, where in the vast majority of cases there is rupture of a plaque, leading to emboli causing cardiac myocyte
ischemia and death.
nuclei were smaller and more numerous in late gestation (embryonic day 18.5) compared to control offspring.
Bers, "Calcium fluxes involved in control of cardiac myocyte
contraction," Circulation Research, vol.
Decreased sarco(endo)plasmic reticulum (SR) [Ca.sup.2+]-uptake and decreased expression of the SR [Ca.sup.2+]-ATPase, SERCA2a, are key features of cardiac myocyte
dysfunction in both experimental and human heartfailure (8,9).
In cardiac pathology, the role of cardiac myocyte
apoptosis is still speculative.
cell cycle control in development, disease, and regeneration.