sup], Ca [sup]2+ is a central player in the excitation-contraction coupling of cardiac myocytes
that keeps heart to contract and relax.
Hopx and Hdac2 interact to modulate Gata4 acetylation and embryonic cardiac myocyte
2+] conductance to suppress early afterdepolarizations in cardiac myocytes
," The Journal of Physiology, vol.
2]) combined with hypoxia protects neonatal cardiac myocyte
but not cardiac fibroblast from hypoxic injury, and apoptosis decreased in human ES-derived cardiac myocytes
hypertrophy with enlarged and hyperchromatic nuclei (hematoxylin-eosin, original magnification x100).
Nitric oxide-dependent parasympathetic signaling is due to activation of constitutive endothelial (type III) nitric oxide synthase in cardiac myocytes
Cyclic GMP signaling and regulation of SERCA activity during cardiac myocyte
Klf15, in turn, controls the level of a potassium channel-interacting protein (KChIP2), which affects how potassium flows out of heart muscle cells called cardiac myocytes
Signaling pathways mediating cardiac myocyte
gene expression in physiological and stress responses.
Tumor necrosis factor-alpha confers resistance to hypoxic injury in the adult mammalian cardiac myocyte
Cellular localization of alpha3betal integrin isoforms in association with myofibrillogenesis during cardiac myocyte
development in culture.
These 21 articles help readers understand the ravages of CHF and the successes and failures of optimal medical therapies and give the basic science behind stem cell research, including the different stem cell types (including hematopoietic, mesenchymal, multipotent adult progenitor cells, umbilical cord blood stem cells, endogenous cardiac stem cells and embryonic stem cells) and the critical physiological pathways including chemokine and homing factor expression, stem cell differentiation toward a cardiac myocyte
phenotype and mechanisms of arrhythmia including electrical coupling.
Estrogen therapy abolished the increase in cardiac myocyte
cross-sectional that was present in the remodeled myocardium of the placebo-treated rats (306 [+ or -] 12 [micro][m.
Ventricular hypertrophy is a process involving cardiac myocyte
hypertrophy and cardiac fibroblast proliferation that results in a structural rearrangement of ventricular chamber and wall components.
Therefore, myocardial infarction not only results in the release of cardiac proteins such as the biomarker troponin but also induces the release of cardiac myocyte