Previous studies  have pointed out that apoptosis within the ischemic penumbra may occur after several days, while necrosis starts in a few hours after the onset of acute brain ischemia
in the ischemic core.
Tymianski, "Emerging mechanisms of disrupted cellular signaling in brain ischemia
," Nature Neuroscience, vol.
Although the role of CysLTs in brain ischemia
is supported by several evidences, the mechanisms through they mediate neuronal injury are not fully clarified.
Susceptibility to focal and glogal brain ischemia
of alzheimer mice displaying a[beta] deposits: effect of immunoglobulin.
Animal experiments demonstrated the ability of PTX to decrease the severity of ischemic/reperfusion brain injury and spontaneous brain ischemia
in spontaneously hypertensive stroke-prone rats (Eun, Liu, 2000; McCarty et al., 2016).
Global Markets Direct's, 'Brain Ischemia
- Pipeline Review, H1 2016', provides an overview of the Brain Ischemia
 It is very well known that brain ischemia
persisting for more than 4 to 6 h will produce permanent neurological damage.
Warach, "Early blood-brain barrier disruption in human focal brain ischemia
," Annals of Neurology, vol.
Detection of free radicals during brain ischemia
and reperfusion by spin trapping and microdialysis.
Important differential diagnoses of cervicocephalic arterial dissection include other vascular or neurological causes of the head and neck pain and/or local neurological syndromes (such as subarachnoid hemorrhage, migraine, and cluster headache) and other causes of brain ischemia
such as cardiac emboli, atherosclerosis, and vasculopathy of brain vessels (14).
1 patient showed arcuate field defects, 1had inferior altitudinal defect 4 Brain infarcts most common 3 patients showed homonymous vessel involved was MCA and hemianopic field defects PCA 5 Brain ischemia
2 showed homonymous hemianopic field defects,
, as a local interruption of blood flow in the brain, is becoming the most common death cause in developed countries; while the transient global ischemia, caused usually by cardiopulmonary arrest, is a more significant cause of cerebral stroke today.
Before discussing a deductive approach to biomedicine, we illustrate the prevailing inductive biomedical approach using the field of brain ischemia
as an example.
In conclusion, functional disorders after brain ischemia
are mainly due to the damage caused by oxidant production in brain cells.
It has been supposed that green tea polyphenols (GTPs) have neuroprotective effects on brain damage after brain ischemia
in animal experiments.