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  • noun

Words related to atheroma

a fatty deposit in the intima (inner lining) of an artery

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Novel risk factors for human carotid atherosclerotic plaques include shear stress, carotid atherosclerotic plaque morphologic risk factors, biochemical risk factors in carotid atherosclerotic plaques and blood, and infection risk factors (such as Chlamydia pneumoniae ).
The carotid color ultrasonography was used to evaluate atherosclerotic plaque morphology and measure intima-media thickness.
The virtue of ultrasound is that it emits sound waves that can detect whether atherosclerotic plaque is "soft" or "hard.
It has been shown that manipulating expression of P4H[alpha]1 affects atherosclerotic plaque stabilization, while different manipulations of P4H[alpha]1 have different influences on lesional collagen synthesis [11-13].
Considering the proinflammatory and prothrombotic properties of plaque necrosis [50], long-term inhibition of apoptosis may not be a favorable strategy to improve atherosclerotic plaque stability.
These systems use a diamond-coated crown, attached to an orbiting shaft, which targets atherosclerotic plaque while preserving healthy vessel tissue, a critical factor in preventing reoccurrences.
31-33) Regardless of the effects of these traditional risk factors on the development of the atherosclerotic plaque, a growing body of evidence demonstrates their impact on rupture-induced occlusion.
Atherosclerotic plaque is the primary, underlying cause of heart disease and stroke in industrialised countries.
Once atherosclerotic plaque has developed, it can become unstable and rupture, which causes blood clot formation in the blood vessel at the site of the rupture," say; Dr.
a biotech company focused on the research and development of compounds to regress atherosclerotic plaque, has released financial results for fiscal 2011.
4] The atherosclerotic plaque contains large numbers of inflammatory cells that may release hydrolytic enzymes and cytokines, thereby destabilizing the plaque (1).
We tested the hypothesis that bacteria from the mouth and/or the gut could end up in the atherosclerotic plaque and thus contribute to the development of cardiovascular disease.
Acute ST-elevation myocardial infarction (AMI) results mostly from acute thrombotic occlusion of an epicardial coronary artery, typically after disruption or erosion of an atherosclerotic plaque and exposure of thrombogenic material to circulating blood.
Using a 17-segment model of the coronary arteries to assess for the presence of calcific or noncalcific plaque, the investigators calculated a total plaque score by summing the number of coronary segments with visible atherosclerotic plaque.
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