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  • adj

Synonyms for proinflammatory

tending to cause inflammation

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References in periodicals archive ?
Evidence supporting the role of proinflammatory genes and oxidative stress in alcoholic brain damage is found by studying drugs that block neurodegeneration.
Increased production of proinflammatory cytokines, such as interleikin (IL)-1, tumor necrosis factor (TNF)-[alpha], by peripheral and heart infiltrating T cells of patients with rheumatic fever and CRHD have been reported in previous studies (6-12).
1-3) The oral mucosal responses to P gingivalis and its LPS are manifested by a marked increase in epithelial cell apoptosis and proinflammatory inter-leukin production, enhancement in nitric oxide and prostaglandin production, and the disturbances in NF[kappa]B (nuclear factor [kappa]B) and MAPK (mitogen-activated protein kinase) signaling cascades.
Although a greater production of pro-inflammatory cytokines at a wound site is beneficial because they will aid healing, 'greater systemic production of proinflammatory cytokines can represent a maladapative response'.
Proinflammatory cytokines (eg, TNF-[alpha], IL-6) released by HIV-infected immunocytes, or by the adipocytes themselves when exposed to these HIV-infected cells, may lead to increased apoptosis (programmed cell death).
Research specific to elevated production of proinflammatory cytokines may provide a new approach to the treatment of pain related to disc degeneration and herniation.
During a study examining mitogen-induced proinflammatory cytokine production, we discovered that 5-mL glass sodium heparin Vacutainer Tubes (lot no.
If you lack the proper amount of Omega 3s in your cells, your body becomes proinflammatory, which means your body is more prone to injury and will heal at a slower rate.
Proinflammatory cytokines play an important role in the pathophysiology of Inflammatory Bowel Disease (IBD).
This peptide-nucleic acid, which to date has shown a very favorable safety profile, appears to stimulate the proinflammatory responses required to combat viral infections such as HIV and human papillomavirus and to dampen aberrant autoimmune-type inflammatory responses, such as occur in patients with rheumatoid arthritis.
The results demonstrate that cellular response to toxic leachables follows a distinct pathway, starting with up-regulation (elevated production) of proinflammatory cytokines and, depending on the strength of the response, terminating in extensive necrosis (localized death of living cells).
Stressful episodes can stimulate your body to produce proinflammatory cytokines, substances that help regulate your immune system.
In the inducible form, which is calcium-calmodulin-independent, activation by proinflammatory cytokines and endotoxins results in up to a 1,000-fold increase in NO production compared with the eNOS form.
After incubations with various oxygen tensions, cells were immediately processed for western blotting, or immunohistochemistry for the presence of various proinflammatory cytokines.
They neutralize and detach microbial cells and their toxic proinflammatory debris from biosurfaces.