Delayed biliary-to-bowel transit in cholescintigraphy after cholecystokinin
Feed intolerance in critical illness is associated with increased basal and nutrient-stimulated plasma cholecystokinin
Role of cholecystokinin
in the motivational control of instrumental action in rats.
A simple enzymatic procedure for radioimmunochemical quantitation of the large molecular forms of gastrin and cholecystokinin
A requiem for the cholecystokinin
Gastrointestinal hormones, such as gastrin, neurotensin, cholecystokinin
and pancreatic polypeptide, are also increased and result in vagal stimulation of smooth muscle fibres.
Exercise has a prokinetic effect on the gut and increases cholecystokinin
levels which stimulates gallbladder contractility and prevents bile stasis.
46-48) Subsequently, a variety of peptide hormones were shown by immunohistochemistry in ampullary carcinoids, including gastrin (associated with Zollinger-Ellison syndrome or multiple endocrine neoplasia type 1 syndrome), vasoactive intestinal peptide, pancreatic polypeptide, corticotropin (associated with Cushing syndrome), insulin, cholecystokinin
, and calcitonin.
production increases in the elderly, and the higher levels may contribute to a number of physiological changes including the slowing of gastric emptying, suppression of appetite and reduction of food intake.
At the gastrointestinal tract level, the presence of food in the stomach is indicated (and relayed to the brain) by signals such as stomach distension and the release of gut hormones such as cholecystokinin
(CCK) and glucagon-like peptide (GLP1).
and hepatic enzymes, cholesterol and lipoproteins in ammonium perfluorooctanoate production workers.
Modulation of spindle oscillations by acetylcholine, cholecystokinin
and 1S,3R-ACPD in the ferret lateral geniculate and perigeniculate nuclei in vitro.
1994) Lack of satiety effect of cholecystokinin
(CCK) in a new rat model not expressing the CCK-A receptor gene.
Expression of cholecystokinin
in the duodenum of patients with coeliac disease: respective role of atrophy and lymphocytic infiltration.
Research has shown that the nocebo negative effect is due to the activation of a molecule involved in anticipation of anxiety called cholecystokinin