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Related to alveolar-capillary membrane: blood-gas barrier
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The increased surface area will amplify the gas liquid interphase at the alveolar-capillary membrane and theoretically the gases should be able to follow naturally occurring pressure gradients with greater efficacy.
In permeability pulmonary edema, pathophysiological disruption of the alveolar-capillary membrane is the basis for the exudation of fluid into the pulmonary interstitium and ultimately into the lungs.
The fibrosis and scarring thicken the alveolar-capillary membrane and impede the diffusion of oxygen from the alveoli into the pulmonary capillary blood.
NPPV reduces the work of the respiratory muscles, decreases respiratory rate, increases tidal volume, and enables gas exchange across the alveolar-capillary membrane.
Once oxygen molecules passively diffuse across the alveolar-capillary membrane, they are transported by the blood to the cells of the tissues.
Assuming a normal alveolar-capillary membrane, as long as four liters per minute of alveolar ventilation match five liters per minute of pulmonary capillary perfusion, gas exchange will be normal.
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