adrenergic


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Related to adrenergic: adrenergic blocking agents, anticholinergic
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  • noun
  • adj

Synonyms for adrenergic

drug that has the effects of epinephrine

relating to epinephrine (its release or action)

References in periodicals archive ?
The action of adrenergic agents on the cardiovascular system may be potentiated by monoamine oxidase inhibitors or tricyclic antidepressants or other drugs known to prolong the QTc interval.
INV102 (nadolol), a beta blocker (beta adrenergic inverse agonist) currently used to treat high blood pressure and migraine, is being repurposed to treat chronic inflammatory airway diseases, including asthma and chronic obstructive pulmonary disease (COPD).
is an alpha-2 adrenergic receptor agonist that acts rapidly and can last for up to 12 hours.
This is the first study describing the genotype and allele frequency of [beta]1 adrenergic receptor gene polymorphisms in any of the Indian population.
2] adrenergic receptors for their clinical relevance.
It has both a positive impact on adrenergic receptors and unique hormonal effects.
This report highlights the importance of a systematic approach to the evaluation of anisocoria and sensitises physicians to the phenomenon of potentiation of the beta adrenergic mydriatic effect of the commonly used nebulisers.
A major target of the adrenergic system is the hippocampus, a region that is critical for learning and memory.
TIMOPTIC (timolol maleate) is a non-selective beta- adrenergic receptor blocking agent indicated for the treatment of elevated intraocular pressure in patients with ocular hypertension or open-angle glaucoma.
Immune cell types associated with innate immunity such as natural killer cells, neutrophils, and macrophages are the potential subjects to be regulated by catecholamines because these cells express functional, [beta]2- and/or [alpha]- adrenergic receptors [17].
MAINTENANCE OF BETA ADRENERGIC RECEPTOR SIGNALING IN HUMAN MICROVASCULAR RETINAL ENDOTHELIAL CELLS.
The investment was triggered by the excellent progress made both by HTL's in-house team and by its founding scientists at the MRC Laboratory of Molecular Biology, Cambridge, who have recently solved the structure of the beta-1 adrenergic receptor based on their novel GPCR-stabilisation methodology.
The main objective of our study was to evaluate the effects of long-term adrenergic beta-blockade on these parameters.
Connor (University of Connecticut Health Center) and Meltzer (University of Massachusetts Medical School) review the pharmacodynamics, indications, dosage, metabolism, and interactions of the stimulants, antidepressants, mood stabilizers, antipsychotics, anxiolytics, and adrenergic agents sometimes prescribed to children and adolescents.