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Glial fibrillary acidic protein in astrocytes in the human neocortex.
The Alexander disease-causing glial fibrillary acidic protein mutant, R416W, accumulates into Rosenthal fibers by a pathway that involves filament aggregation and the association of alpha B-crystallin and HSP27.
Immunohistochemical results for antigen tests using vimentin, neuron-specific enolase (NSE), SI00, and glial fibrillary acidic protein (GFAP) in a 5-year-old, female golden eagle with tetraplegia.
The multifocal immunolabeling of neoplastic cells for neuron-specific enolase, synaptophysin, glial fibrillary acidic protein, and adrenocorticotrophic hormone appeared, whereas a few cells reacted with vimentin and S-100, and stained negative for other markers, which were also used for case 1.
Determination of S-100 and glial fibrillary acidic protein concentrations in cerebrospinal fluid after brain infarction.
2002), we evaluated the effects of CPF exposure on glial fibrillary acidic protein (GFAP), an astrocyte-associated protein (Garcia et al.
The sections were stained for S-100 protein, glial fibrillary acidic protein (GFAP), cytokeratin, and microtubule-associated protein.
Expression of glial fibrillary acidic protein (GFAP) in peripheral nerve sheath tumors: a comparative study of immunoreactivity of GFAP, vimentin, S-100 protein, and neurofilament in 38 schwannomas and 18 neurofibromas.
Glial fibrillary acidic protein (GFAP) is a monomeric filament protein specific to the brain astrocytes (89).
Ethanol exposure affects glial fibrillary acidic protein gene expression and transcription during rat brain development.
Rare cases may stain positively for desmin and glial fibrillary acidic protein, but not typically for leukocyte common antigen or actin.
Immunostains with glial fibrillary acidic protein, melanoma marker, epithelial membrane antigen, and carcinoembryonic antigen were negative.
The proteins S100B, neuron-specific enolase (NSE) [8] myelin basic protein, and the recently introduced glial fibrillary acidic protein are currently being evaluated as protein markers in cerebrospinal fluid and/or blood for detection of brain injury in neurologic patients (1-3) and patients with cerebrovascular accidents (4), traumatic brain injury (5), stroke (6), global cerebral ischemia attributable to cardiac arrest or cardiopulmonary bypass surgery (7, 8), tumor cerebri (9), or dementia (10), with the aim to eventually locate the site of injury (neuron, glia, or myelin).
It's remarkable to consider how far we've come since 1978 when one considers that for his job interviews, Bernd spoke about the latest development of the time, glial fibrillary acidic protein immunohistochemistry.