rumen

(redirected from Forestomach)
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  • noun

Synonyms for rumen

the first compartment of the stomach of a ruminant

References in periodicals archive ?
Protozoa in the forestomach of the collared peccary (Tayassu tajacu).
This effect was noted for several digestive tract cancers, specifically cancers of the esophagus and the nonglandular forestomach (5) (Doll et al.
Lam has inhibited tumors in the lung, skin, and forestomach of mice with limonoids.
sativum and are able to suppress BaP-induced neoplasia in the mouse forestomach.
Additional studies using laboratory mice showed that green tea has the property of preventing chemically induced cancers, including that of the lung, forestomach, esophagus, duodenum, pancreas, liver and colon.
So far, animals given carcinogens and green tea--or EGCG in water--have developed fewer tumors of the skin, lung, esophagus, forestomach, small intestine, colon, liver, pancreas, and breast.
Mesynthes's matrix is derived from the propria-submucosa layers of the ovine (sheep) forestomach, a tissue that is very similar to skin.
Effects of BHA and related phenols on the forestomach of rats.
The in vivo and in vitro inhibitory effects of TQ against 20-methylcholanthrene (MC)-induced fibrosarcoma (Badary and Gamal, 2001) and against benzo[a]pyrene-induced forestomach carcinogenesis (Badary et al.
It is known that the forestomach of all ruminants plays a primary role in assimilation of nitrogen-containing substances.
CLA has also been found to inhibit tumor initiation of mouse forestomach carcinogenesis [22].
Over the past quarter century, dozens of animal studies have demonstrated the ability of phenolic antioxidants -- notably BHT and BHA -- to inhibit chemically induced cancers in the lung, liver, forestomach, skin, breast and colon.
Fistula Feeder: Veterinarians, studying how bovine innards work, install a hole, called a fistula, into a cow's rumen, the 30-gallon forestomach, where microbes ferment grass.
It has been reported that disruption of Keapl in mice leads to hyperkeratosis in esophagus, forestomach, and skin, most likely because of constitutive activation of NRF2 and aberrant expression of some ARE-dependent cytokeratins (Wakabayashi et al.