Cox-2 inhibitor

(redirected from Cyclooxygenase-2 inhibitor)
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Words related to Cox-2 inhibitor

an anti-inflammatory drug that fights pain and blocks Cox-2 activity without impeding the activity of Cox-1

References in periodicals archive ?
The risk of cardiovascular thrombotic events with selective cyclooxygenase-2 inhibitors.
The select cyclooxygenase-2 inhibitor celecoxib reduced the extent of atherosclerosis in apo E-/- mice.
Physicians should avoid nonselective NSAIDs and cyclooxygenase-2 inhibitors in patients with renal or liver disease.
The FDA cited concerns with cardiovascular safety when it issued a "not approvable" letter for Merck's cyclooxygenase-2 inhibitor etoricoxib (Arcoxia) for the same indication in April.
Amsterdam -- Extended-release acetaminophen is a possible alternative to cyclooxygenase-2 inhibitors for pain associated with knee osteoarthritis, Dr.
Now that the future availability of at least one selective cyclooxygenase-2 inhibitor appears certain, physicians will need better guidance on how to weigh risks and benefits--and avoid liability--when treating individual patients.
HONOLULU -- Rofecoxib, a selective cyclooxygenase-2 inhibitor, significantly reduced the frequency of perimenstrual migraine headaches in a small, open-label trial.
Giesler gave 50 mg of the cyclooxygenase-2 inhibitor rofecoxib (Vioxx) on the morning of surgery to 13 patients.
Now that the future availability of at least one selective cyclooxygenase-2 inhibitor appears certain, guidance on how physicians can weigh risks and benefits--and avoid liability--when treating their individual patients is clearly inadequate.
Cyclooxygenase-2 inhibitors in tumorigenesis (Part I).
At that meeting, the panelists also voted 16-9 that there were not enough data to suggest that naproxen presented a substantially lower risk of CV events than did either ibuprofen or selective NSAIDs, such as cyclooxygenase-2 inhibitors.
The meeting panelists also voted 16-9 that there were not enough data to suggest that naproxen presented a substantially lower risk of CV events than did either ibuprofen or selective NSAIDs, such as cyclooxygenase-2 inhibitors.
Selective cyclooxygenase-2 inhibitors show a differential ability to inhibit proliferation and induce apoptosis of colon adenocarcinoma cells.
The cyclooxygenase-2 inhibitors did not convey a significant increase in myocardial infarctions, stroke, cardiovascular death, or other thrombotic cardiovascular adverse events when compared with non-steroidal anti-inflammatory drugs.
M was receiving, but also other, often-overlooked medications, including nonsteroidal anti-inflammatory drugs (NSAIDs), cyclooxygenase-2 inhibitors, calcium channel blockers, and systemic corticosteroids.
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