Comparison of renal toxicity after long-term oral administration of cadmium chloride
and cadmium-metallothionein in rats.
The groups included the control and cadmium chloride
groups (15 and 25 [micro]M/kg).
has been classified a human carcinogen.
Table 1: Comparative Representation of Toxicity Indices for Arsenic Trioxide and Cadmium Chloride
with and without PFT-[alpha] in Rat Liver Cells and Neoplastic HepG2 Cell Line.
2002) observed a decline in liver and ovarian proteins of Notopterus notopterus after exposure to various concentrations of cadmium chloride
and mercuric chloride, individually and in combination, for 2 months.
was used in all studies that involved cadmium exposure.
Experiments were performed in control and metallothionein-null mice, injected subcutaneously with a wide range of cadmium chloride
doses, six times per week for up to 10 wk.
Oral exposure to cadmium chloride
triggers an acute inflammatory response in the intestines of mice, initiated bythe over-expression of tissue macrophage inflammatory protein-2 mRNA.
Comparative efficacy of two microdoses of a potentized homeopathic drug, Cadmium sulphoricum, in reducing genotoxic effects produced by cadmium chloride
in mice: a time course study.
Moreover, the eukaryotic translation initiation factor 4E gene, a cellular target for toxicity and death caused by exposure to cadmium chloride
(Othumpangat et al.
Costa Mesa, CA, USA); recombinant murine IL-4 from PeproTech (Rocky Hill, NJ, USA); and analytical-grade nickel sulfate, aluminum chloride, strontium chloride, cadmium chloride
, and lead nitrate from POCH (Gliwice, Poland).
As a reference, 20 [micro]M cadmium chloride
Expression of hsp 27, hsp 60, hsc 70 and hsp 76 by immortalized human proximal tubule cells (HK-2) following exposure to heat shock, sodium arsenite or cadmium chloride
Applications of gene arrays in environmental toxicology: Rngerprints of gene regulation associated with cadmium chloride
, benzo(a)pyrene, and trichloroethylene.
Involvement of the extracellular signal-regulated protein kinase (ERK) pathway in the induction of apoptosis by cadmium chloride
in CCRF-CEM cells.